Use of cardiac troponin T, creatine kinase and its isoform to monitor myocardial injury during radiofrequency ablation for supraventricular tachycardia

Kou Gi Shyu, Jiunn Lee Lin, Jin Jer Chen, Hang Chang

研究成果: 雜誌貢獻文章同行評審

17 引文 斯高帕斯(Scopus)

摘要

To determine whether radiofrequency ablation for supraventricular tachycardia causes significant minor myocardial injury, 16 patients with supraventricular tachycardia undergoing radiofrequency ablation were studied. Cardiac troponin T, creatine kinase and its MB form (CKMB) were measured before, immediately after ablation and every 6 h thereafter for 24 h to detect myocardial injury. Elevation of creatine kinase, CKMB and cardiac troponin T was observed in 6, 4 and 5 patients, respectively. The peak mean creatine kinase concentration was 167 ± 152 IU/l and that of CKMB was 9 ± 6 IU/l. The peak mean cardiac troponin T level was 0.44 ± 0.47 ng/ml. The frequency of elevated measurements was not statistically different among creatine kinase, CKMB and cardiac troponin T. The mean pulse numbers of ablation, mean duration of ablation, radiofrequency current and mean total energy did not differ statistically between those with or without elevated cardiac troponin T. It was concluded that radiofrequency ablation for supraventricular tachycardia indeed caused some minor myocardial injury and the frequency of elevated cardiac troponin T was comparable to that of CKMB.
原文英語
頁(從 - 到)392-395
頁數4
期刊Cardiology
87
發行號5
出版狀態已發佈 - 9月 1996
對外發佈

ASJC Scopus subject areas

  • 心臟病學與心血管醫學

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