Pathophysiology of cancer therapy-provoked atrial fibrillation

Wan Li Cheng, Yu Hsun Kao, Shih Ann Chen, Yi Jen Chen

研究成果: 雜誌貢獻文章

5 引文 (Scopus)

摘要

Atrial fibrillation (AF) occurs with increased frequency in cancer patients, especially in patients who undergo surgery or chemotherapy. AF disturbs the prognosis of cancer patients and challenges therapeutic outcomes of cancer treatment. Elucidating the mechanisms of cancer-induced AF would help identify specific strategies for preventing AF occurrence. In addition to concurrent risk factors of cancer and AF, cancer surgery, side effects of anticancer agents, and cancer-associated immune responses play critical roles in the genesis of AF. In this review, we provide succinct potential mechanisms of AF genesis in cancer patients.

原文英語
頁(從 - 到)186-194
頁數9
期刊International Journal of Cardiology
219
DOIs
出版狀態已發佈 - 九月 15 2016

指紋

Atrial Fibrillation
Neoplasms
Therapeutics
Antineoplastic Agents
Drug Therapy

ASJC Scopus subject areas

  • Cardiology and Cardiovascular Medicine

引用此文

Pathophysiology of cancer therapy-provoked atrial fibrillation. / Cheng, Wan Li; Kao, Yu Hsun; Chen, Shih Ann; Chen, Yi Jen.

於: International Journal of Cardiology, 卷 219, 15.09.2016, p. 186-194.

研究成果: 雜誌貢獻文章

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abstract = "Atrial fibrillation (AF) occurs with increased frequency in cancer patients, especially in patients who undergo surgery or chemotherapy. AF disturbs the prognosis of cancer patients and challenges therapeutic outcomes of cancer treatment. Elucidating the mechanisms of cancer-induced AF would help identify specific strategies for preventing AF occurrence. In addition to concurrent risk factors of cancer and AF, cancer surgery, side effects of anticancer agents, and cancer-associated immune responses play critical roles in the genesis of AF. In this review, we provide succinct potential mechanisms of AF genesis in cancer patients.",
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AB - Atrial fibrillation (AF) occurs with increased frequency in cancer patients, especially in patients who undergo surgery or chemotherapy. AF disturbs the prognosis of cancer patients and challenges therapeutic outcomes of cancer treatment. Elucidating the mechanisms of cancer-induced AF would help identify specific strategies for preventing AF occurrence. In addition to concurrent risk factors of cancer and AF, cancer surgery, side effects of anticancer agents, and cancer-associated immune responses play critical roles in the genesis of AF. In this review, we provide succinct potential mechanisms of AF genesis in cancer patients.

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