Mechanism of fulminant pulmonary edema caused by enterovirus 71

Shang Jyh Kao, Fwu Lin Yang, Yung Hsiang Ksu, Hsing I. Chen

研究成果: 雜誌貢獻文章

74 引文 斯高帕斯(Scopus)

摘要

Pulmonary edema (PE) may occur with enterovirus 71 (EV71) infection. We monitored arterial pressure (AP) and heart rate (HR) in patients with EV71 infection and analyzed the variability of AP and HR. Sympathetic activity, AP, and HR increased with respiratory stress. Thereafter, parasympathetic activity increased with decreases in AP and HR. The lungs showed edema with inducible nitric oxide synthase (iNOS) expression. Destruction of the medial, ventral, and caudal medulla may lead to sympathetic overactivation, causing blood to shift to the lungs. The pathogenesis of PE may also involve iNOS and nitric oxide.
原文英語
頁(從 - 到)1784-1788
頁數5
期刊Clinical Infectious Diseases
38
發行號12
DOIs
出版狀態已發佈 - 六月 15 2004

ASJC Scopus subject areas

  • Immunology

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    Kao, S. J., Yang, F. L., Ksu, Y. H., & Chen, H. I. (2004). Mechanism of fulminant pulmonary edema caused by enterovirus 71. Clinical Infectious Diseases, 38(12), 1784-1788. https://doi.org/10.1086/421021