Electrophysiological mechanisms and catheter ablation of complex atrial arrhythmias from crista terminalis: Insight from three-dimensional noncontact mapping

Yenn Jiang Lin, Ching Tai Tai, Tu Ying Liu, Satoshi Higa, Pi Chang Lee, Jin Long Huang, Yoga Yuniadi, Bien Hsien Huang, Kun Tai Lee, Shih Huang Lee, Kuang Chang Ueng, Ming Hsung Hsieh, Yu An Ding, Shih Ann Chen

研究成果: 雜誌貢獻文章

22 引文 (Scopus)

摘要

Paroxysmal atrial fibrillation (PAF) can be initiated by ectopic activation from the crista terminalis. The crista terminalis conduction gap is also a critical isthmus in atrial reentrant arrhythmias like upper and lower loop reentry. The aim of this study was to investigate the mechanism and results of catheter ablation for complex atrial arrhythmias originating from the crista terminalis using the noncontact mapping system (NCM). The study population consisted of six patients (5 men, 1 woman; 70 ± 9 years) with drug refractory PAF and typical/atypical atrial flutter. NCM identified the earliest ectopic activation originating from the crista terminalis in these six patients. The reentry circuit of atypical atrial flutter propagated around the upper crista terminalis in five patients, and lower crista terminalis in one patient. The reentry circuit of atypical atrial flutter and the initial reentry circuit of AF conducted through the crista terminalis gap in all patients. Radiofrequency applications were delivered on the sites of ectopy, which initiated AF. Substrate modification was also performed over the crista terminalis gap (six patients) and cavotricuspid isthmus (three patients) responsible for the reentry. During a mean follow-up of 9 ± 5 months (range 5-18 months), five patients were free of AF without antiarrhythmic drugs, and one patient did not have AF or atrial flutter using propafenone. NCM demonstrated the mechanism of crista terminalis ectopy-initiating AF and associated typical/atypical atrial flutter. Catheter ablation of crista terminalis ectopy and substrate for the reentry guided by NCM successfully eliminated these atrial arrhythmias.
原文英語
頁(從 - 到)1231-1239
頁數9
期刊PACE - Pacing and Clinical Electrophysiology
27
發行號9
DOIs
出版狀態已發佈 - 九月 2004
對外發佈Yes

指紋

Catheter Ablation
Cardiac Arrhythmias
Atrial Flutter
Atrial Fibrillation
Propafenone
Anti-Arrhythmia Agents

ASJC Scopus subject areas

  • Cardiology and Cardiovascular Medicine

引用此文

Electrophysiological mechanisms and catheter ablation of complex atrial arrhythmias from crista terminalis : Insight from three-dimensional noncontact mapping. / Lin, Yenn Jiang; Tai, Ching Tai; Liu, Tu Ying; Higa, Satoshi; Lee, Pi Chang; Huang, Jin Long; Yuniadi, Yoga; Huang, Bien Hsien; Lee, Kun Tai; Lee, Shih Huang; Ueng, Kuang Chang; Hsieh, Ming Hsung; Ding, Yu An; Chen, Shih Ann.

於: PACE - Pacing and Clinical Electrophysiology, 卷 27, 編號 9, 09.2004, p. 1231-1239.

研究成果: 雜誌貢獻文章

Lin, Yenn Jiang ; Tai, Ching Tai ; Liu, Tu Ying ; Higa, Satoshi ; Lee, Pi Chang ; Huang, Jin Long ; Yuniadi, Yoga ; Huang, Bien Hsien ; Lee, Kun Tai ; Lee, Shih Huang ; Ueng, Kuang Chang ; Hsieh, Ming Hsung ; Ding, Yu An ; Chen, Shih Ann. / Electrophysiological mechanisms and catheter ablation of complex atrial arrhythmias from crista terminalis : Insight from three-dimensional noncontact mapping. 於: PACE - Pacing and Clinical Electrophysiology. 2004 ; 卷 27, 編號 9. 頁 1231-1239.
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abstract = "Paroxysmal atrial fibrillation (PAF) can be initiated by ectopic activation from the crista terminalis. The crista terminalis conduction gap is also a critical isthmus in atrial reentrant arrhythmias like upper and lower loop reentry. The aim of this study was to investigate the mechanism and results of catheter ablation for complex atrial arrhythmias originating from the crista terminalis using the noncontact mapping system (NCM). The study population consisted of six patients (5 men, 1 woman; 70 ± 9 years) with drug refractory PAF and typical/atypical atrial flutter. NCM identified the earliest ectopic activation originating from the crista terminalis in these six patients. The reentry circuit of atypical atrial flutter propagated around the upper crista terminalis in five patients, and lower crista terminalis in one patient. The reentry circuit of atypical atrial flutter and the initial reentry circuit of AF conducted through the crista terminalis gap in all patients. Radiofrequency applications were delivered on the sites of ectopy, which initiated AF. Substrate modification was also performed over the crista terminalis gap (six patients) and cavotricuspid isthmus (three patients) responsible for the reentry. During a mean follow-up of 9 ± 5 months (range 5-18 months), five patients were free of AF without antiarrhythmic drugs, and one patient did not have AF or atrial flutter using propafenone. NCM demonstrated the mechanism of crista terminalis ectopy-initiating AF and associated typical/atypical atrial flutter. Catheter ablation of crista terminalis ectopy and substrate for the reentry guided by NCM successfully eliminated these atrial arrhythmias.",
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T2 - Insight from three-dimensional noncontact mapping

AU - Lin, Yenn Jiang

AU - Tai, Ching Tai

AU - Liu, Tu Ying

AU - Higa, Satoshi

AU - Lee, Pi Chang

AU - Huang, Jin Long

AU - Yuniadi, Yoga

AU - Huang, Bien Hsien

AU - Lee, Kun Tai

AU - Lee, Shih Huang

AU - Ueng, Kuang Chang

AU - Hsieh, Ming Hsung

AU - Ding, Yu An

AU - Chen, Shih Ann

PY - 2004/9

Y1 - 2004/9

N2 - Paroxysmal atrial fibrillation (PAF) can be initiated by ectopic activation from the crista terminalis. The crista terminalis conduction gap is also a critical isthmus in atrial reentrant arrhythmias like upper and lower loop reentry. The aim of this study was to investigate the mechanism and results of catheter ablation for complex atrial arrhythmias originating from the crista terminalis using the noncontact mapping system (NCM). The study population consisted of six patients (5 men, 1 woman; 70 ± 9 years) with drug refractory PAF and typical/atypical atrial flutter. NCM identified the earliest ectopic activation originating from the crista terminalis in these six patients. The reentry circuit of atypical atrial flutter propagated around the upper crista terminalis in five patients, and lower crista terminalis in one patient. The reentry circuit of atypical atrial flutter and the initial reentry circuit of AF conducted through the crista terminalis gap in all patients. Radiofrequency applications were delivered on the sites of ectopy, which initiated AF. Substrate modification was also performed over the crista terminalis gap (six patients) and cavotricuspid isthmus (three patients) responsible for the reentry. During a mean follow-up of 9 ± 5 months (range 5-18 months), five patients were free of AF without antiarrhythmic drugs, and one patient did not have AF or atrial flutter using propafenone. NCM demonstrated the mechanism of crista terminalis ectopy-initiating AF and associated typical/atypical atrial flutter. Catheter ablation of crista terminalis ectopy and substrate for the reentry guided by NCM successfully eliminated these atrial arrhythmias.

AB - Paroxysmal atrial fibrillation (PAF) can be initiated by ectopic activation from the crista terminalis. The crista terminalis conduction gap is also a critical isthmus in atrial reentrant arrhythmias like upper and lower loop reentry. The aim of this study was to investigate the mechanism and results of catheter ablation for complex atrial arrhythmias originating from the crista terminalis using the noncontact mapping system (NCM). The study population consisted of six patients (5 men, 1 woman; 70 ± 9 years) with drug refractory PAF and typical/atypical atrial flutter. NCM identified the earliest ectopic activation originating from the crista terminalis in these six patients. The reentry circuit of atypical atrial flutter propagated around the upper crista terminalis in five patients, and lower crista terminalis in one patient. The reentry circuit of atypical atrial flutter and the initial reentry circuit of AF conducted through the crista terminalis gap in all patients. Radiofrequency applications were delivered on the sites of ectopy, which initiated AF. Substrate modification was also performed over the crista terminalis gap (six patients) and cavotricuspid isthmus (three patients) responsible for the reentry. During a mean follow-up of 9 ± 5 months (range 5-18 months), five patients were free of AF without antiarrhythmic drugs, and one patient did not have AF or atrial flutter using propafenone. NCM demonstrated the mechanism of crista terminalis ectopy-initiating AF and associated typical/atypical atrial flutter. Catheter ablation of crista terminalis ectopy and substrate for the reentry guided by NCM successfully eliminated these atrial arrhythmias.

KW - Ablation

KW - Atrial fibrillation

KW - Crista terminalis

KW - Mapping

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