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Both PKA and Epac Pathways Mediate N-Acetylcysteine-Induced Connexin43 Preservation in Rats with Myocardial Infarction
Tsung-Ming Lee, Shinn Zong Lin,
Nen Chung Chang
內科學科
心臟血管內科
臺北醫學大學附設醫院
臺北醫學大學臺北心臟醫學研究中心
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引文 斯高帕斯(Scopus)
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深入研究「Both PKA and Epac Pathways Mediate N-Acetylcysteine-Induced Connexin43 Preservation in Rats with Myocardial Infarction」主題。共同形成了獨特的指紋。
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Agriculture & Biology
myocardial infarction
100%
acetylcysteine
98%
cAMP-dependent protein kinase
90%
rats
39%
brefeldin A
29%
infarction
28%
arrhythmia
27%
proteins
25%
ex vivo studies
17%
tau-protein kinase
17%
lithium chloride
14%
gap junctions
13%
coronary vessels
13%
myocytes
10%
Western blotting
9%
dyes
8%
inactivation
8%
reverse transcriptase polymerase chain reaction
8%
Medicine & Life Sciences
Connexin 43
92%
Acetylcysteine
85%
Cyclic AMP-Dependent Protein Kinases
78%
Myocardial Infarction
55%
Brefeldin A
29%
N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide
29%
Proteins
27%
Cardiac Arrhythmias
18%
Infarction
18%
Lithium Chloride
15%
Glycogen Synthase Kinase 3
14%
Gap Junctions
12%
Protein Kinase Inhibitors
11%
Muscle Cells
9%
Wistar Rats
8%
Coloring Agents
8%
Coronary Vessels
8%
Real-Time Polymerase Chain Reaction
8%
Western Blotting
7%
Chemical Compounds
Protein
31%
Brefeldin A
30%
Arrhythmic
18%
Cyclic Amp Dependent Protein Kinase Inhibitor
17%
Antiarrhythmic Agent
13%
Western Blotting
12%
Lithium Chloride
11%
Glycogen
11%
Dye
5%