MOESM7 of Enrichment of superoxide dismutase 2 in glioblastoma confers to acquisition of temozolomide resistance that is associated with tumor-initiating cell subsets

  • Chia Hung Chien (Contributor)
  • Jian-Ying Chuang (Contributor)
  • Shung-Tai Yang (Contributor)
  • Wen-Bin Yang (Contributor)
  • Pin Yuan Chen (Contributor)
  • Tsung-I Hsu (Contributor)
  • Chih Yuan Huang (Contributor)
  • Wei-Lun Lo (Contributor)
  • Ka-Yen Yang (Contributor)
  • Ming Sheng Liu (Contributor)
  • Jui Mei Chu (Contributor)
  • Pei Hsuan Chung (Contributor)
  • Jr Jiun Liu (Contributor)
  • Shao Wen Chou (Contributor)
  • Shang Hung Chen (Contributor)
  • Kwang Yu Chang (Contributor)



Additional file 7: Figure S7. The analysis from clinical samples. (A) The IHC image represented a GBM tissue section which was stained with anti-rabbit IgG as a negative control. (B) The IHC staining in the left was a representative result of SOD2 and Bmi1 from a same patient. The study was done in total of the clinical specimens from 10 patients with recurrent GBM. The area of the staining was assessed and recorded in the dotted graph at right, showing statistical correlation of SOD2 and Bmi1.
可用日期1月 1 2019
發行者Unknown Publisher